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This notion would be consistent with recent studies which demonstrate that air pollution, particularly PM 2.5 , can alter mtDNA, presumably though oxidative damage [10,11,12], and that prenatal air pollution exposure increases cord blood concentrations of a mitochondrial-derived peptides that are associated with long-term mitochondrial metabolism alterations [13]
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Mitochondrial fission is primarily regulated by the dynamin-related protein 1 (Drp-1) in humans [236]._Mitochondrial dynamics are vital for retaining a balanced level of mitochondrial ROS, buffering the number of dysfunctional units, and avoiding the initiation of a positive loop of ROS and damaged mtDNA [167, 237, 238]
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