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TB-500’s key mechanisms include: Actin sequestration: By binding G-actin (monomeric actin), TB-500 keeps it available for cell migration and wound repair rather than polymerizing prematurely into structural filaments

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[DOI] [PubMed] [Google Scholar] 180.Lee JY, Choo JE, Park HJ, Park JB, Lee SC, Jo I, et al