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In this context, studies on uraemic rats have demonstrated that elevated extracellular phosphate levels drive vascular calcification by upregulating NOX4 and its p22 subunit, leading to increased ROS production (Table 2) [17,18]
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In vivo studies using a murine model of TNBC showed that AuPhos-89 significantly inhibits tumor growth, positioning it as a promissory compound for cancer treatment [128]
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