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Uric Acid as a Redox Switch in Gout: Linking Xanthine Oxidoreductase-Derived ROS, NLRP3 Inflammasome Activation and Emerging Ferroptotic Mechanisms
The discussion that follows is applicable to xenobiotics in general (including drugs) and to some extent to endogenous compounds
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When these fibers become overactive or malfunction, they send more pain signals to the spinal cord, increasing central sensitization
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