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The inhibition of NQO1 by dicoumarol was suggested to stimulate formation of superoxide, overt oxidative stress, subsequent suppress of cell growth, and induce apoptosis in pancreatic cancer cells [35,36]

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Knockdown of METTL14 by siRNA significantly reduces GPX4 mRNA m^6A modification, restores GPX4 expression, alleviates ECM degradation, and ultimately inhibits chondrocyte ferroptosis in vitro (176)