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Oxidative damage promotes the release of pro-inflammatory factors such as IL-1β and tumor necrosis factor α through activation of NLRP3 inflammatory vesicles and the NF-κB pathway, 7,69,70 while the inflammatory microenvironment further induces ROS generation, which leads to lipid peroxidation of the meibomian glands, destroying the structure of the lipid layer and accelerating tear evaporation
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