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This mechanistic duality dictates that therapeutic interventions targeting amino acid metabolism must be rigorously calibrated to the precise tissue context and metabolic dosage
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ROS also activate microglia and astrocytes via p38MAPK/NF-κB and JAK2/STAT3 pathways, inducing pro-inflammatory cytokine production and disrupting the neuronal microenvironment, exacerbating OS and cell death (Linnerbauer et al., 2020
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