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This could imply that GSTP1 associated with cellular membranes is catalytically inactive and plays a different role at this location (e.g., a signaling rather than catalytic function)

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Under normal conditions, 98% or more of GSH is maintained in its reduced state, however, in oxidative stress-associated disorders like cancer and neurodegenerative diseases, an appreciable amount of the GSH pool exists in the oxidized state, GSSG