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Constitutively active form of GPCR increases the chance of phosphorylation by G protein-coupled receptor kinases (GRKs) or other kinases (cAMP-dependent PKA and lipid signaling-activated PKC), and facilitates the binding of β -arrestin to the phosphorylated receptor, blocking G protein interaction with a receptor [22–24]

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10.1038/224117a0 [DOI] [PubMed] [Google Scholar] 15.Ballatori N, Krance SM, Notenboom S, Shi S, Tieu K, Hammond CL