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Despite a certain degree of individual variability in the symptomatology and neurological profile of the disease, pathological commonalities across all types of TBI comprise excitotoxicity, ionic disturbances, decreased cerebral blood flow, oedema, oxidative stress, inflammation, and damage to and death of brain cells that may occur during acute (i.e., within minutes) and subacute phases (i.e., within 24 h) [8], and potentially continuing for months or decades after the injury [9]