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[20] Although not a routine method for estimating the time since deposition (TSD) of bloodstains, targeted molecular analyses used in TSD research – such as proteomics, metabolomics, and nucleic-acid profiling – may incorporate immunoaffinity-based enrichment steps to improve sensitivity for low-abundance biomarkers

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Studies in human hepatoma cells have shown that GSTP1 is able to bind to signal transduction and transcriptional activator 3 (STAT3) to form the GSTP1-STAT3 complex, inhibiting epidermal growth factor (EGF)-mediated phosphorylation of STAT3 tyrosine and preventing its transcriptional activity, thereby reducing cell proliferation and halting the cell cycle [73]