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reported the induction of tolerogenic mouse DCs (with the capacity to induce regulatory T cells) by stimuli like small spleen peptides, IL-10, or transforming growth factor beta (TGF-β) to be mediated by mTOR signaling ( In contrast to the mainly glycolytic phenotype described above, a different metabolic phenotype is observed for M2-macrophages that are involved in resolution of inflammation, tissue repair and wound healing, but also tissue remodeling and induction of Th2 responses via the secretion of immune-suppressive cytokines like IL-10 and TGF-β, the Th2 cytokine IL-13, but also other cytokines and factors promoting angiogenesis [reviewed in ( Figure 2 )